File:Nematode C. elegans Stages of wild type spermatogenesis.jpg

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Description Figure 5. (A) Stages of wild type spermatogenesis are shown diagramatically as an ordered pathway of morphogenesis with the stages labeled in blue. Some of the > 44 known genes are placed on the pathway where ultrastructural or light microscopic defects are evident. The ife-1 gene is not shown, but it slows down the rate of spermatogenesis and also affects spermatozoa. The next to last step of the pathway represents mutant spermatozoa that are cytologically normal but cannot enter oocytes. The spe-11 mutant is a paternal effect lethal mutant that makes spermatozoa that enter the egg and the resulting defective embryo dies. (B) The most common terminal stages of mutants that arrest morphogenesis without forming spermatids. (C) The protein distribution of SPE-9 (arrows), the hypothesized SPE-9 receptor (“Y” on egg surface) and the SPE-41/TRP-3 protein (red dots). Please note that individual drawings are not to scale; for instance, spermatozoa are 5–6 μm in diameter while the eggs shown are ~45 μm (modified from L'Hernault et al., 1988).
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Source http://www.wormbook.org/chapters/www_spermatogenesis/spermatogenesis.html L'Hernault, S.W. Spermatogenesis (February 20, 2006), WormBook, ed. The C. elegans Research Community, WormBook, doi/10.1895/wormbook.1.85.1, http://www.wormbook.org.
Author L'Hernault, S.W.

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current15:28, 27 February 2024Thumbnail for version as of 15:28, 27 February 20241,793 × 724 (522 KB)Rasbak (talk | contribs){{Information |description=Figure 5. (A) Stages of wild type spermatogenesis are shown diagramatically as an ordered pathway of morphogenesis with the stages labeled in blue. Some of the > 44 known genes are placed on the pathway where ultrastructural or light microscopic defects are evident. The ife-1 gene is not shown, but it slows down the rate of spermatogenesis and also affects spermatozoa. The next to last step of the pathway represents mutant spermatozoa that are cytologically normal b...

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