File:The diseases of infancy and childhood (1910) (14577357259).jpg

Identifier: diseasesofinfa00kopl (find matches)
Title: The diseases of infancy and childhood
Year: 1910 (1910s)
Authors: Koplik, Henry, 1858- (from old catalog)
Subjects: Children
Publisher: New York and Philadelphia, Lea & Febiger
Contributing Library: The Library of Congress
Digitizing Sponsor: The Library of Congress

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erior basic meningitis is socalled because the inflammation affects the posterior part of the baseof the brain and the structures in this location, and rarely spreads tothe vertex of the brain, at most only affecting the tips of the temporo-sphenoidal lobes, and in some cases extending forward to the opticcommissure. These cases were first described by Gee and Barlow,in the Bartholomew Hospital Reports of 1878, and subsequently byStill in 1898. Occurrence.—The affection occurs in infants and children belowthe age of two years, and is rarely seen in older children. I have seenexceptional cases in children of three and five years of age. Etiology.—These cases, according to Still, and confirmed by myown observations, are caused by a diplococcus which is identical withthe diplococcus of Weichselbaum, Jager, and Leichtenstern, an intra-cellular diplococcus not staining with Grams method. AlthoughStill thought that these were only sporadic cases of the epidemic form PLATE XXI / / i j\ H
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Posterior Basic Meningitis. (Gee, Barlow, and Still.Authors ease. MENINGITIS. 365 of cerebrospinal meningitis, it can now be said that they are seeD veryfrequently and in large numbers in epidemics of cerebrospinal menin-gitis, and may occur sporadically. They are only specific, inasmuchas they are a form of cerebrospinal meningitis as it occurs in youngerchildren and infants. These cases divide themselves into those which are fatal after sixweeks; those which die after three or four months with hydroceph-alus ; and those which recover. In the first set of cases anatomicallywe find pus and lymph at the base of the brain and extending downthe cord. In the second set of cases there is simply thickening of thepia and arachnoid, with adhesions between the cerebellum and me-dulla. The inflammation may spread down the cord to a varyingdegree and upward along the lining membrane of the ventricles, andafterward along the base as far as the optic commissure. In thechronic cases there may be ad

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