File:Studies in cardiac pathology (1911) (14778476544).jpg

Identifier: studiesincardiac00norr (find matches)
Title: Studies in cardiac pathology
Year: 1911 (1910s)
Authors: Norris, George William, 1875-1965
Subjects: Cardiovascular System
Publisher: Philadelphia : Saunders
Contributing Library: Columbia University Libraries
Digitizing Sponsor: Open Knowledge Commons

View Book Page: Book Viewer
About This Book: Catalog Entry
View All Images: All Images From Book
Click here to view book online to see this illustration in context in a browseable online version of this book.

Text Appearing Before Image:
rent degree of the cardiac lesion. At the PennsylvaniaHospital there were 6 cases in 1,300 autopsies; at the PhiladelphiaHospital, 28 among 8,640. Cardiac rupture may occur as the result of fatty degeneration,aneurismal dilatation, suppurative myocarditis, acute necrosis,gummatous disease, and brown atrophy. This condition wasfirst described by Harvey, and later by Morgagni. AmongQuains 100 collected cases 77 were due to fatty degeneration.Most of the patients were beyond sixt.y years of age, and suddendeath occurred in 71 per cent. The site of rupture was as follows:Left ventricle, 55; right ventricle, 7; right auricle, 3; left auricle, 2.Rupture may be complete or incomplete. It is slightly morefrequent in males. In 18 of LetuUes 110 cases, multiple rupturewas found. Occasionally rupture occurs in apparently healthyyoung people as the result of coronary thrombosis.^ Quainscases included two between the ages of ten and twenty. Rupture ■ Klingmann: New York Med. Jour., 190S, p. 199.
Text Appearing After Image:
Fig. 69.—Gumma of the Myocardium. A larfcc, firm, nodular mass is seen in the septum below the aortic valves which bulgesforward into the ventricle. Another mass is seen in the wall of the ventricle. These masseswere firm, .slightly elastic, grayish in color, and upon microscopic examination showed typicalgummatous structure. The heart is that of a patient aged thirty-one years, who had contracted syphilis eightyears before. Three years previous to admission he had been incapacitated for three daysby an inflammation of the heart. Six weeks before admission he had had fugacious painsand less of power in the right hand. A few days afterward he had a syncopal attack while atwork, following which he developed headache, weakness, numbness of the legs, and diplopia.On admi.s.sion to the hospital he was found to have a right-sided hemiplegia. On auscultationa mitral systolic murmur was noted. During his three months stay in the hospital he hadhemoptysis and cardiac pain. (Pennsylvania Ho.s

Note About Images